دانلود رایگان مقاله انگلیسی منجر شدن کم کاری غده پارتیروئید به تشنج - هینداوی 2018

A 24-year-old man presented to the Chris Hani Baragwanath Academic Hospital emergency department with recurrent seizures having previously been diagnosed with epilepsy from age 14. ,e biochemical investigations and brain imaging were suggestive of seizures secondary to hypocalcemia, and a diagnosis of idiopathic hypoparathyroidism was confirmed. After calcium and vitamin D replacement, the patient recovered well and is seizure free, and off antiepileptic therapy. ,is case highlights the occurrence of brain calcinosis in idiopathic hypoparathyroidism; the occurrence of acute symptomatic seizures due to provoking factors other than epilepsy; and the importance, in the correct clinical setting, of considering alternative, and sometimes treatable, causes of seizures other than epilepsy.

4. Conclusion

This case highlights acute symptomatic seizures, which are caused by disease processes other than epilepsy, and highlights the need, in the correct clinical setting, to consider other treatable differential diagnosis for seizures. In this patient, despite a prior diagnosis of epilepsy, he now remains seizure free and off antiepileptic drugs. Furthermore, we highlight the entity of brain calcinosis in hypoparathyroidism, a diagnosis that should be considered when these radiological features are seen on a brain CT scan.